Matrix Metalloproteinase-9, Neuron-specific Enolase, S100 B and Tau Protein Levels in the Patients with Carbon monoxide Poisoning

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dc.authorscopusid57196010964en_US
dc.authorscopusid24337141100en_US
dc.authorscopusid24824246400en_US
dc.authorscopusid57196003969en_US
dc.authorscopusid55999287400en_US
dc.authorscopusid26433450000en_US
dc.authorscopusid57193566202en_US
dc.contributor.authorCan, B.
dc.contributor.authorZengin, S.
dc.contributor.authorEren, Ş.H.
dc.contributor.authorBoğan, M.
dc.contributor.authorSabak, M.
dc.contributor.authorGülen, B.
dc.contributor.authorUlusal, H.
dc.date.accessioned2024-08-23T16:07:29Z
dc.date.available2024-08-23T16:07:29Z
dc.date.issued2023en_US
dc.departmentDüzce Üniversitesien_US
dc.description.abstractBackground: S100B, NSE, MMP-9, and Tau protein levels increase in cases causing hypoxic cell damage. The diagnosis of the severity of carbon monoxide (CO) poisoning in the early period of these parameters was studied. Material and Methods: COHb level measurement was made using a signal capture CO-pulse oximeter (Masimo's SET Rainbow, Masimo's Co, USA) at the first admission of the patients. Then, COHb levels were confirmed by arterial blood gas(ABG) analysis. The patients were divided into two groups as mild and moderate-severe, according to their Glasgow coma scores (GCS) [Mild (14–15); Moderate (9–13) or Severe (3–8)]. The control group was composed of 16 healthy and non-smoking volunteers. Results: The serum S100B protein and MMP-9 values at 0 hr of admission in the hospital and 3hr of treatment were not significantly different in the patient group as compared to the control group. Tau protein levels were significantly higher in the patient group at 0 and 3 hours (p> 0.05) as compared to healthy person. Conclusion: There was no relationship between CO poisoning and MMP-9 and S100B protein levels. NSE and Tau protein were significantly higher in the patient group than the control group. Tau protein may be more useful marker as compared to neuron-specific enolase. © 2023 The Author(s). Published by Bentham Science Publisher.en_US
dc.description.sponsorshipFirat University Scientific Research Projects Management Unit, FÜBAP, (TF.UT.16.02)en_US
dc.identifier.doi10.2174/0250688204666230508115135
dc.identifier.issn0250-6882
dc.identifier.issue2en_US
dc.identifier.scopus2-s2.0-85167567172en_US
dc.identifier.scopusqualityQ4en_US
dc.identifier.urihttps://doi.org/10.2174/0250688204666230508115135
dc.identifier.urihttps://hdl.handle.net/20.500.12684/14680
dc.identifier.volume4en_US
dc.indekslendigikaynakScopusen_US
dc.language.isoenen_US
dc.publisherBentham Science Publishersen_US
dc.relation.ispartofNew Emirates Medical Journalen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectAcute carbon monoxideen_US
dc.subjectCarbon monoxide poisoningen_US
dc.subjectHypoxic cell damageen_US
dc.subjectMatrix metalloproteinase 9en_US
dc.subjectPatientsen_US
dc.subjectTau proteinen_US
dc.titleMatrix Metalloproteinase-9, Neuron-specific Enolase, S100 B and Tau Protein Levels in the Patients with Carbon monoxide Poisoningen_US
dc.typeArticleen_US

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